CEBPA (P49715) variants and mutations

CEBPA (also known as P49715) is a human protein-coding gene encoding a CCAAT/enhancer-binding protein alpha protein. It drives granulocytic differentiation and helps maintain normal myeloid maturation. Acquired pathogenic variants define important subsets of acute myeloid leukemia, while germline variants can confer familial AML predisposition. This analysis covers 1,467 CEBPA variants and mutations. Of these, 90% have computational variant effect predictions. Disease context includes acute myeloid leukemia, hereditary neoplastic syndrome, and Inherited cancer-predisposing syndrome. Example CEBPA variants include E2D, E2G, and E2K.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, ClinGen, MaveDB, LitVar.

Notable CEBPA variants

Examples include E2D, E2G, E2K, S3L, A4D, A4G, A4S, A4T. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.