BBS2 (BBSome complex member BBS2) variants and mutations

BBS2 (also known as BBSome complex member BBS2) is a human protein-coding gene encoding a BBSome complex member protein. It contributes to BBSome assembly and ciliary cargo trafficking, which are required for signaling in photoreceptors, kidney, hypothalamus, and other tissues. Biallelic loss-of-function variants cause Bardet-Biedl syndrome. This analysis covers 1,104 BBS2 variants and mutations. Of these, 79% have computational variant effect predictions. Disease context includes Bardet-Biedl syndrome, Bardet-Biedl syndrome 2, and retinitis pigmentosa. Example BBS2 variants include M1V, L2M, and L3P.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, AlphaFold DB, gnomAD constraint, Open Targets, MaveDB, LitVar.

Notable BBS2 variants

Examples include M1V, L2M, L3P, L3Q, P4L, V5M, F6L, T7I. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.