ATP7B (Copper-transporting ATPase 2) variants and mutations

ATP7B (also known as Copper-transporting ATPase 2) is a human protein-coding gene encoding a copper-transporting ATPase 2 protein. A copper-transporting ATPase that moves excess copper out of cells and helps deliver it from liver cells into bile. Its trafficking between intracellular membranes is central to copper homeostasis, and ATP7B dysfunction causes Wilson disease. This analysis covers 2,584 ATP7B variants and mutations. Of these, 96% have computational variant effect predictions. Disease context includes Wilson disease, genetic disorder, and Dystonia. Example ATP7B variants include M1I, E3D, and Q4E.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, ClinGen, PharmGKB, MaveDB, LitVar.

Notable ATP7B variants

Examples include M1I, E3D, Q4E, Q4H, Q4P, Q4R, E5*, E5K. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.