X-linked lymphoproliferative disease due to SH2D1A deficiency: genes and variants

X-linked lymphoproliferative disease due to SH2D1A deficiency is linked to 1 analyzed protein (SH2D1A). 7 DNA variants are known to cause it; 24 more are uncertain, and 1 of those already look disease-causing on computable evidence.

Last updated 2026-09-30. Research information, not medical advice.

Genes linked to X-linked lymphoproliferative disease due to SH2D1A deficiency

Known disease-causing variants in X-linked lymphoproliferative disease due to SH2D1A deficiency

VariantPositionProtein partClinical label
SH2D1A M1T1Disease-causing (★★)
SH2D1A D2G2Disease-causing (★★)
SH2D1A M1V1Disease-causing (★)
SH2D1A I84T84SH2Disease-causing (★)
SH2D1A Y54N54SH2Disease-causing (★)
SH2D1A R32T32SH2Disease-causing
SH2D1A R55L55SH2Disease-causing

Uncertain variants in X-linked lymphoproliferative disease due to SH2D1A deficiency that look disease-causing

VariantPositionProtein partClinical labelEvidence
SH2D1A R55Q55SH2Conflicting reports (★)+7: 2 other pathogenic changes within 3 positions; R55L at the same position is pathogenic; seen in 0 of gnomAD DNA copies; REVEL 0.869

Diseases related to X-linked lymphoproliferative disease due to SH2D1A deficiency

Frequently asked questions

Which genes are linked to X-linked lymphoproliferative disease due to SH2D1A deficiency?

In CATVariant, X-linked lymphoproliferative disease due to SH2D1A deficiency is linked to 1 analyzed protein: SH2D1A (SH2 domain-containing protein 1A).

How many genetic variants are linked to X-linked lymphoproliferative disease due to SH2D1A deficiency?

31 variants: 7 are classified as disease-causing (pathogenic or likely pathogenic) in ClinVar and 24 are of uncertain significance or have conflicting reports.

Which uncertain variants in X-linked lymphoproliferative disease due to SH2D1A deficiency look disease-causing?

1 uncertain variants reach the likely-pathogenic range of the ACMG/AMP points scale on computable evidence, for example SH2D1A R55Q. These are leads for expert review, not diagnoses.

About this data

Variant–disease links come from ClinVar, Open Targets and UniProt, pooled from the latest public CATVariant analysis of each human protein. Evidence scores use the ACMG/AMP Bayesian points scale with computable criteria only (position among known disease variants, rarity in gnomAD, calibrated predictors, deep mutational scanning); there is no family or patient data, so they prioritise variants for expert review and never classify them.

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