Werner syndrome: genes and variants

Werner syndrome is linked to 1 analyzed protein (WRN). 7 DNA variants are known to cause it; 1,671 more are uncertain, and 2 of those already look disease-causing on computable evidence.

Last updated 2026-09-30. Research information, not medical advice.

Genes linked to Werner syndrome

Where Werner syndrome variants cluster

Known disease-causing variants in Werner syndrome

VariantPositionProtein partClinical label
WRN G574R574Helicase ATP-bindingDisease-causing (★★)
WRN S707G707Helicase ATP-bindingDisease-causing (★★)
WRN K37N37Interaction with WRNIP1Disease-causing (★)
WRN K577M577Helicase ATP-bindingDisease-causing
WRN K125N1253'-5' exonucleaseDisease-causing
WRN K135E1353'-5' exonucleaseDisease-causing
WRN Q1229L1229HRDCDisease-causing

Uncertain variants in Werner syndrome that look disease-causing

VariantPositionProtein partClinical labelEvidence
WRN G574E574Helicase ATP-bindingUncertain (★)+7: 2 other pathogenic changes within 3 positions; G574R at the same position is pathogenic; seen in 6.6e-06 of gnomAD DNA copies; REVEL 0.773
WRN G574V574Helicase ATP-bindingUncertain (★)+6: 2 other pathogenic changes within 3 positions; G574R at the same position is pathogenic; seen in 2.8e-06 of gnomAD DNA copies; REVEL 0.760

Diseases related to Werner syndrome

Frequently asked questions

Which genes are linked to Werner syndrome?

In CATVariant, Werner syndrome is linked to 1 analyzed protein: WRN (Bifunctional 3'-5' exonuclease/ATP-dependent helicase WRN).

How many genetic variants are linked to Werner syndrome?

1,779 variants: 7 are classified as disease-causing (pathogenic or likely pathogenic) in ClinVar and 1,671 are of uncertain significance or have conflicting reports.

Which uncertain variants in Werner syndrome look disease-causing?

2 uncertain variants reach the likely-pathogenic range of the ACMG/AMP points scale on computable evidence, for example WRN G574E and WRN G574V. These are leads for expert review, not diagnoses.

About this data

Variant–disease links come from ClinVar, Open Targets and UniProt, pooled from the latest public CATVariant analysis of each human protein. Evidence scores use the ACMG/AMP Bayesian points scale with computable criteria only (position among known disease variants, rarity in gnomAD, calibrated predictors, deep mutational scanning); there is no family or patient data, so they prioritise variants for expert review and never classify them.

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