Thyroid adenoma, hyperfunctioning, somatic: genes and variants

Thyroid adenoma, hyperfunctioning, somatic is linked to 1 analyzed protein (TSHR). 5 DNA variants are known to cause it; 0 more are uncertain, and 0 of those already look disease-causing on computable evidence.

Last updated 2026-09-30. Research information, not medical advice.

Genes linked to Thyroid adenoma, hyperfunctioning, somatic

Known disease-causing variants in Thyroid adenoma, hyperfunctioning, somatic

VariantPositionProtein partClinical label
TSHR D619G619CytoplasmicDisease-causing
TSHR L629F629TransmembraneDisease-causing
TSHR F631L631TransmembraneDisease-causing
TSHR S281I281ExtracellularDisease-causing
TSHR A623I623CytoplasmicDisease-causing

Same protein, different disease

Diseases related to Thyroid adenoma, hyperfunctioning, somatic

Frequently asked questions

Which genes are linked to Thyroid adenoma, hyperfunctioning, somatic?

In CATVariant, Thyroid adenoma, hyperfunctioning, somatic is linked to 1 analyzed protein: TSHR (Thyrotropin receptor).

How many genetic variants are linked to Thyroid adenoma, hyperfunctioning, somatic?

5 variants: 5 are classified as disease-causing (pathogenic or likely pathogenic) in ClinVar and 0 are of uncertain significance or have conflicting reports.

Which uncertain variants in Thyroid adenoma, hyperfunctioning, somatic look disease-causing?

None of the uncertain variants currently reaches the likely-pathogenic range on computable evidence alone.

About this data

Variant–disease links come from ClinVar, Open Targets and UniProt, pooled from the latest public CATVariant analysis of each human protein. Evidence scores use the ACMG/AMP Bayesian points scale with computable criteria only (position among known disease variants, rarity in gnomAD, calibrated predictors, deep mutational scanning); there is no family or patient data, so they prioritise variants for expert review and never classify them.

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