Syndromic X-linked intellectual disability Najm type: genes and variants

Syndromic X-linked intellectual disability Najm type is linked to 1 analyzed protein (CASK). 8 DNA variants are known to cause it; 17 more are uncertain, and 0 of those already look disease-causing on computable evidence.

Last updated 2026-09-30. Research information, not medical advice.

Genes linked to Syndromic X-linked intellectual disability Najm type

Known disease-causing variants in Syndromic X-linked intellectual disability Najm type

VariantPositionProtein partClinical label
CASK L209P209Protein kinaseDisease-causing (★★)
CASK R255C255Protein kinaseDisease-causing (★★)
CASK R489W489PDZDisease-causing (★★)
CASK G659D659SH3Disease-causing (★)
CASK V854A854Guanylate kinase-likeDisease-causing (★)
CASK H120R120Protein kinaseDisease-causing (★)
CASK V719M719Required for interaction with NRXN1 (via C-termiDisease-causing (★)
CASK S810N810Guanylate kinase-likeDisease-causing (★)

Same protein, different disease

Diseases related to Syndromic X-linked intellectual disability Najm type

Frequently asked questions

Which genes are linked to Syndromic X-linked intellectual disability Najm type?

In CATVariant, Syndromic X-linked intellectual disability Najm type is linked to 1 analyzed protein: CASK (Peripheral plasma membrane protein CASK).

How many genetic variants are linked to Syndromic X-linked intellectual disability Najm type?

54 variants: 8 are classified as disease-causing (pathogenic or likely pathogenic) in ClinVar and 17 are of uncertain significance or have conflicting reports.

Which uncertain variants in Syndromic X-linked intellectual disability Najm type look disease-causing?

None of the uncertain variants currently reaches the likely-pathogenic range on computable evidence alone.

About this data

Variant–disease links come from ClinVar, Open Targets and UniProt, pooled from the latest public CATVariant analysis of each human protein. Evidence scores use the ACMG/AMP Bayesian points scale with computable criteria only (position among known disease variants, rarity in gnomAD, calibrated predictors, deep mutational scanning); there is no family or patient data, so they prioritise variants for expert review and never classify them.

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