Spastic tetraplegia and axial hypotonia, progressive: genes and variants

Spastic tetraplegia and axial hypotonia, progressive is linked to 1 analyzed protein (SOD1). 5 DNA variants are known to cause it; 2 more are uncertain, and 0 of those already look disease-causing on computable evidence.

Last updated 2026-09-30. Research information, not medical advice.

Genes linked to Spastic tetraplegia and axial hypotonia, progressive

Known disease-causing variants in Spastic tetraplegia and axial hypotonia, progressive

VariantPositionProtein partClinical label
SOD1 I114T114Disease-causing (★★)
SOD1 F21L21Disease-causing (★★)
SOD1 L85F85Disease-causing (★★)
SOD1 G86S86Disease-causing (★★)
SOD1 G42S42Disease-causing (★★)

Same protein, different disease

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Frequently asked questions

Which genes are linked to Spastic tetraplegia and axial hypotonia, progressive?

In CATVariant, Spastic tetraplegia and axial hypotonia, progressive is linked to 1 analyzed protein: SOD1 (Superoxide dismutase [Cu-Zn]).

How many genetic variants are linked to Spastic tetraplegia and axial hypotonia, progressive?

7 variants: 5 are classified as disease-causing (pathogenic or likely pathogenic) in ClinVar and 2 are of uncertain significance or have conflicting reports.

Which uncertain variants in Spastic tetraplegia and axial hypotonia, progressive look disease-causing?

None of the uncertain variants currently reaches the likely-pathogenic range on computable evidence alone.

About this data

Variant–disease links come from ClinVar, Open Targets and UniProt, pooled from the latest public CATVariant analysis of each human protein. Evidence scores use the ACMG/AMP Bayesian points scale with computable criteria only (position among known disease variants, rarity in gnomAD, calibrated predictors, deep mutational scanning); there is no family or patient data, so they prioritise variants for expert review and never classify them.

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