Short stature and advanced bone age, with or without early-onset osteoarthritis and/or osteochondritis dissecans: genes and variants

Short stature and advanced bone age, with or without early-onset osteoarthritis and/or osteochondritis dissecans is linked to 1 analyzed protein (ACAN). 4 DNA variants are known to cause it; 14 more are uncertain, and 0 of those already look disease-causing on computable evidence.

Last updated 2026-09-30. Research information, not medical advice.

Genes linked to Short stature and advanced bone age, with or without early-onset osteoarthritis and/or osteochondritis dissecans

Known disease-causing variants in Short stature and advanced bone age, with or without early-onset osteoarthritis and/or osteochondritis dissecans

VariantPositionProtein partClinical label
ACAN D2381N2381C-type lectinDisease-causing (★★)
ACAN L302P302Link 2Disease-causing (★)
ACAN V2417M2417C-type lectinDisease-causing (★)
ACAN L2355P2355C-type lectinDisease-causing

Diseases related to Short stature and advanced bone age, with or without early-onset osteoarthritis and/or osteochondritis dissecans

Frequently asked questions

Which genes are linked to Short stature and advanced bone age, with or without early-onset osteoarthritis and/or osteochondritis dissecans?

In CATVariant, Short stature and advanced bone age, with or without early-onset osteoarthritis and/or osteochondritis dissecans is linked to 1 analyzed protein: ACAN (Aggrecan core protein).

How many genetic variants are linked to Short stature and advanced bone age, with or without early-onset osteoarthritis and/or osteochondritis dissecans?

28 variants: 4 are classified as disease-causing (pathogenic or likely pathogenic) in ClinVar and 14 are of uncertain significance or have conflicting reports.

Which uncertain variants in Short stature and advanced bone age, with or without early-onset osteoarthritis and/or osteochondritis dissecans look disease-causing?

None of the uncertain variants currently reaches the likely-pathogenic range on computable evidence alone.

About this data

Variant–disease links come from ClinVar, Open Targets and UniProt, pooled from the latest public CATVariant analysis of each human protein. Evidence scores use the ACMG/AMP Bayesian points scale with computable criteria only (position among known disease variants, rarity in gnomAD, calibrated predictors, deep mutational scanning); there is no family or patient data, so they prioritise variants for expert review and never classify them.

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