Severe intellectual disability-progressive spastic diplegia syndrome: genes and variants

Severe intellectual disability-progressive spastic diplegia syndrome is linked to 1 analyzed protein (CTNNB1). 5 DNA variants are known to cause it; 13 more are uncertain, and 0 of those already look disease-causing on computable evidence.

Last updated 2026-09-30. Research information, not medical advice.

Genes linked to Severe intellectual disability-progressive spastic diplegia syndrome

Known disease-causing variants in Severe intellectual disability-progressive spastic diplegia syndrome

VariantPositionProtein partClinical label
CTNNB1 S348F348ARM 5Disease-causing (★)
CTNNB1 L424R424ARM 7Disease-causing (★)
CTNNB1 G575R575Disease-causing (★)
CTNNB1 E692D692Disease-causing (★)
CTNNB1 G216R216ARM 2Disease-causing

Same protein, different disease

Diseases related to Severe intellectual disability-progressive spastic diplegia syndrome

Frequently asked questions

Which genes are linked to Severe intellectual disability-progressive spastic diplegia syndrome?

In CATVariant, Severe intellectual disability-progressive spastic diplegia syndrome is linked to 1 analyzed protein: CTNNB1 (Catenin beta-1).

How many genetic variants are linked to Severe intellectual disability-progressive spastic diplegia syndrome?

44 variants: 5 are classified as disease-causing (pathogenic or likely pathogenic) in ClinVar and 13 are of uncertain significance or have conflicting reports.

Which uncertain variants in Severe intellectual disability-progressive spastic diplegia syndrome look disease-causing?

None of the uncertain variants currently reaches the likely-pathogenic range on computable evidence alone.

About this data

Variant–disease links come from ClinVar, Open Targets and UniProt, pooled from the latest public CATVariant analysis of each human protein. Evidence scores use the ACMG/AMP Bayesian points scale with computable criteria only (position among known disease variants, rarity in gnomAD, calibrated predictors, deep mutational scanning); there is no family or patient data, so they prioritise variants for expert review and never classify them.

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