Prothrombin deficiency: genes and variants

Prothrombin deficiency is linked to 1 analyzed protein (F2). 17 DNA variants are known to cause it; 41 more are uncertain, and 0 of those already look disease-causing on computable evidence.

Last updated 2026-09-30. Research information, not medical advice.

Also known as: congenital prothrombin deficiency

Genes linked to Prothrombin deficiency

Where Prothrombin deficiency variants cluster

Known disease-causing variants in Prothrombin deficiency

VariantPositionProtein partClinical label
F2 R596Q596Peptidase S1Disease-causing (★★)
F2 R461W461Peptidase S1Disease-causing (★★)
F2 R500Q500Peptidase S1Disease-causing (★★)
F2 R581C581Peptidase S1Disease-causing (★★)
F2 R425C425Peptidase S1Disease-causing (★)
F2 R596W596Peptidase S1Disease-causing (★)
F2 V424M424Peptidase S1Disease-causing (★)
F2 G499E499Peptidase S1Disease-causing (★)
F2 G332A332Disease-causing (★)
F2 E357G357Disease-causing (★)
F2 R425H425Peptidase S1Disease-causing
F2 D595E595Peptidase S1Disease-causing
F2 E352K352Disease-causing
F2 E343K343Disease-causing
F2 V365E365Peptidase S1Disease-causing
F2 M380T380Peptidase S1Disease-causing
F2 G601V601Peptidase S1Disease-causing

Diseases related to Prothrombin deficiency

Frequently asked questions

Which genes are linked to Prothrombin deficiency?

In CATVariant, Prothrombin deficiency is linked to 1 analyzed protein: F2 (Prothrombin).

How many genetic variants are linked to Prothrombin deficiency?

75 variants: 17 are classified as disease-causing (pathogenic or likely pathogenic) in ClinVar and 41 are of uncertain significance or have conflicting reports.

Which uncertain variants in Prothrombin deficiency look disease-causing?

None of the uncertain variants currently reaches the likely-pathogenic range on computable evidence alone.

About this data

Variant–disease links come from ClinVar, Open Targets and UniProt, pooled from the latest public CATVariant analysis of each human protein. Evidence scores use the ACMG/AMP Bayesian points scale with computable criteria only (position among known disease variants, rarity in gnomAD, calibrated predictors, deep mutational scanning); there is no family or patient data, so they prioritise variants for expert review and never classify them.

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