Neurodevelopmental disorder with hypotonia, impaired speech, and behavioral abnormalities: genes and variants

Neurodevelopmental disorder with hypotonia, impaired speech, and behavioral abnormalities is linked to 1 analyzed protein (GNAI1). 8 DNA variants are known to cause it; 10 more are uncertain, and 0 of those already look disease-causing on computable evidence.

Last updated 2026-09-30. Research information, not medical advice.

Genes linked to Neurodevelopmental disorder with hypotonia, impaired speech, and behavioral abnormalities

Where Neurodevelopmental disorder with hypotonia, impaired speech, and behavioral abnormalities variants cluster

Known disease-causing variants in Neurodevelopmental disorder with hypotonia, impaired speech, and behavioral abnormalities

VariantPositionProtein partClinical label
GNAI1 T48K48G-alphaDisease-causing (★★)
GNAI1 G45D45G-alphaDisease-causing (★★)
GNAI1 D272N272G-alphaDisease-causing (★★)
GNAI1 D173V173G-alphaDisease-causing (★★)
GNAI1 T48I48G-alphaDisease-causing (★)
GNAI1 G40C40G-alphaDisease-causing (★)
GNAI1 Q204R204G-alphaDisease-causing (★)
GNAI1 D272G272G-alphaDisease-causing

Frequently asked questions

Which genes are linked to Neurodevelopmental disorder with hypotonia, impaired speech, and behavioral abnormalities?

In CATVariant, Neurodevelopmental disorder with hypotonia, impaired speech, and behavioral abnormalities is linked to 1 analyzed protein: GNAI1 (Guanine nucleotide-binding protein G(i) subunit alpha-1).

How many genetic variants are linked to Neurodevelopmental disorder with hypotonia, impaired speech, and behavioral abnormalities?

26 variants: 8 are classified as disease-causing (pathogenic or likely pathogenic) in ClinVar and 10 are of uncertain significance or have conflicting reports.

Which uncertain variants in Neurodevelopmental disorder with hypotonia, impaired speech, and behavioral abnormalities look disease-causing?

None of the uncertain variants currently reaches the likely-pathogenic range on computable evidence alone.

About this data

Variant–disease links come from ClinVar, Open Targets and UniProt, pooled from the latest public CATVariant analysis of each human protein. Evidence scores use the ACMG/AMP Bayesian points scale with computable criteria only (position among known disease variants, rarity in gnomAD, calibrated predictors, deep mutational scanning); there is no family or patient data, so they prioritise variants for expert review and never classify them.

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