Merosin deficient congenital muscular dystrophy: genes and variants

Merosin deficient congenital muscular dystrophy is linked to 1 analyzed protein (LAMA2). 9 DNA variants are known to cause it; 112 more are uncertain, and 0 of those already look disease-causing on computable evidence.

Last updated 2026-09-30. Research information, not medical advice.

Genes linked to Merosin deficient congenital muscular dystrophy

Known disease-causing variants in Merosin deficient congenital muscular dystrophy

VariantPositionProtein partClinical label
LAMA2 C1079R1079Laminin EGF-like 12Disease-causing (★★)
LAMA2 M1T1Disease-causing (★★)
LAMA2 H2627Q2627Laminin G-like 3Disease-causing (★★)
LAMA2 R1508K1508Laminin EGF-like 16Disease-causing (★★)
LAMA2 G284R284Laminin N-terminalDisease-causing (★)
LAMA2 L243P243Laminin N-terminalDisease-causing (★)
LAMA2 T821P821Laminin EGF-like 7Disease-causing
LAMA2 E722K722Laminin IV type A 1Disease-causing
LAMA2 I2761T2761Disease-causing

Which prediction tools work for Merosin deficient congenital muscular dystrophy

How often each tool ranks a disease-causing variant above a harmless one (AUROC × 100).

Same protein, different disease

Diseases related to Merosin deficient congenital muscular dystrophy

Frequently asked questions

Which genes are linked to Merosin deficient congenital muscular dystrophy?

In CATVariant, Merosin deficient congenital muscular dystrophy is linked to 1 analyzed protein: LAMA2 (Laminin subunit alpha-2).

How many genetic variants are linked to Merosin deficient congenital muscular dystrophy?

128 variants: 9 are classified as disease-causing (pathogenic or likely pathogenic) in ClinVar and 112 are of uncertain significance or have conflicting reports.

Which uncertain variants in Merosin deficient congenital muscular dystrophy look disease-causing?

None of the uncertain variants currently reaches the likely-pathogenic range on computable evidence alone.

Which variant effect predictor works best for Merosin deficient congenital muscular dystrophy?

Among tools not trained on clinical labels, SIFT separates this disease's known disease-causing variants from harmless ones best (AUROC 0.68, based on 8 disease-causing and 76 harmless variants).

About this data

Variant–disease links come from ClinVar, Open Targets and UniProt, pooled from the latest public CATVariant analysis of each human protein. Evidence scores use the ACMG/AMP Bayesian points scale with computable criteria only (position among known disease variants, rarity in gnomAD, calibrated predictors, deep mutational scanning); there is no family or patient data, so they prioritise variants for expert review and never classify them.

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