Leydig cell agenesis: genes and variants

Leydig cell agenesis is linked to 1 analyzed protein (LHCGR). 10 DNA variants are known to cause it; 13 more are uncertain, and 0 of those already look disease-causing on computable evidence.

Last updated 2026-09-30. Research information, not medical advice.

Genes linked to Leydig cell agenesis

Where Leydig cell agenesis variants cluster

Known disease-causing variants in Leydig cell agenesis

VariantPositionProtein partClinical label
LHCGR D578G578TransmembraneDisease-causing (★★)
LHCGR S616Y616TransmembraneDisease-causing (★★)
LHCGR E354K354ExtracellularDisease-causing (★)
LHCGR A498V498TransmembraneDisease-causing (★)
LHCGR D578N578TransmembraneDisease-causing (★)
LHCGR C343S343ExtracellularDisease-causing
LHCGR V144F144LRR 2Disease-causing
LHCGR C543R543TransmembraneDisease-causing
LHCGR A593P593TransmembraneDisease-causing
LHCGR L502P502TransmembraneDisease-causing

Which prediction tools work for Leydig cell agenesis

How often each tool ranks a disease-causing variant above a harmless one (AUROC × 100).

Same protein, different disease

Diseases related to Leydig cell agenesis

Frequently asked questions

Which genes are linked to Leydig cell agenesis?

In CATVariant, Leydig cell agenesis is linked to 1 analyzed protein: LHCGR (Lutropin-choriogonadotropic hormone receptor).

How many genetic variants are linked to Leydig cell agenesis?

26 variants: 10 are classified as disease-causing (pathogenic or likely pathogenic) in ClinVar and 13 are of uncertain significance or have conflicting reports.

Which uncertain variants in Leydig cell agenesis look disease-causing?

None of the uncertain variants currently reaches the likely-pathogenic range on computable evidence alone.

Which variant effect predictor works best for Leydig cell agenesis?

Among tools not trained on clinical labels, SIFT separates this disease's known disease-causing variants from harmless ones best (AUROC 0.92, based on 9 disease-causing and 8 harmless variants).

About this data

Variant–disease links come from ClinVar, Open Targets and UniProt, pooled from the latest public CATVariant analysis of each human protein. Evidence scores use the ACMG/AMP Bayesian points scale with computable criteria only (position among known disease variants, rarity in gnomAD, calibrated predictors, deep mutational scanning); there is no family or patient data, so they prioritise variants for expert review and never classify them.

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