LCAT deficiency: genes and variants

LCAT deficiency is linked to 1 analyzed protein (LCAT). 10 DNA variants are known to cause it; 4 more are uncertain, and 0 of those already look disease-causing on computable evidence.

Last updated 2026-09-30. Research information, not medical advice.

Genes linked to LCAT deficiency

Known disease-causing variants in LCAT deficiency

VariantPositionProtein partClinical label
LCAT R123C123Disease-causing (★★)
LCAT V333M333Disease-causing (★)
LCAT M404V404Disease-causing (★)
LCAT L233P233Disease-causing
LCAT M276K276Disease-causing
LCAT M317I317Disease-causing
LCAT W170R170Disease-causing
LCAT N252K252Disease-causing
LCAT A117T117Disease-causing
LCAT R182C182Disease-causing

Which prediction tools work for LCAT deficiency

How often each tool ranks a disease-causing variant above a harmless one (AUROC × 100).

Same protein, different disease

Diseases related to LCAT deficiency

Frequently asked questions

Which genes are linked to LCAT deficiency?

In CATVariant, LCAT deficiency is linked to 1 analyzed protein: LCAT (Phosphatidylcholine-sterol acyltransferase).

How many genetic variants are linked to LCAT deficiency?

26 variants: 10 are classified as disease-causing (pathogenic or likely pathogenic) in ClinVar and 4 are of uncertain significance or have conflicting reports.

Which uncertain variants in LCAT deficiency look disease-causing?

None of the uncertain variants currently reaches the likely-pathogenic range on computable evidence alone.

Which variant effect predictor works best for LCAT deficiency?

Among tools not trained on clinical labels, SIFT separates this disease's known disease-causing variants from harmless ones best (AUROC 1.00, based on 10 disease-causing and 8 harmless variants).

About this data

Variant–disease links come from ClinVar, Open Targets and UniProt, pooled from the latest public CATVariant analysis of each human protein. Evidence scores use the ACMG/AMP Bayesian points scale with computable criteria only (position among known disease variants, rarity in gnomAD, calibrated predictors, deep mutational scanning); there is no family or patient data, so they prioritise variants for expert review and never classify them.

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