Ivacaftor response - Efficacy: genes and variants

Ivacaftor response - Efficacy is linked to 1 analyzed protein (CFTR). 4 DNA variants are known to cause it; 0 more are uncertain, and 0 of those already look disease-causing on computable evidence.

Last updated 2026-09-30. Research information, not medical advice.

Genes linked to Ivacaftor response - Efficacy

Known disease-causing variants in Ivacaftor response - Efficacy

VariantPositionProtein partClinical label
CFTR S945L945ABC transmembrane type-1 2Disease-causing (★★★)
CFTR G1244E1244ABC transporter 2Disease-causing (★★★)
CFTR P67L67CytoplasmicDisease-causing (★★★)
CFTR E193K193ABC transmembrane type-1 1Disease-causing (★★★)

Same protein, different disease

Diseases related to Ivacaftor response - Efficacy

Frequently asked questions

Which genes are linked to Ivacaftor response - Efficacy?

In CATVariant, Ivacaftor response - Efficacy is linked to 1 analyzed protein: CFTR (Cystic fibrosis transmembrane conductance regulator).

How many genetic variants are linked to Ivacaftor response - Efficacy?

13 variants: 4 are classified as disease-causing (pathogenic or likely pathogenic) in ClinVar and 0 are of uncertain significance or have conflicting reports.

Which uncertain variants in Ivacaftor response - Efficacy look disease-causing?

None of the uncertain variants currently reaches the likely-pathogenic range on computable evidence alone.

About this data

Variant–disease links come from ClinVar, Open Targets and UniProt, pooled from the latest public CATVariant analysis of each human protein. Evidence scores use the ACMG/AMP Bayesian points scale with computable criteria only (position among known disease variants, rarity in gnomAD, calibrated predictors, deep mutational scanning); there is no family or patient data, so they prioritise variants for expert review and never classify them.

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