Iodotyrosyl coupling defect: genes and variants

Iodotyrosyl coupling defect is linked to 1 analyzed protein (TG). 8 DNA variants are known to cause it; 158 more are uncertain, and 0 of those already look disease-causing on computable evidence.

Last updated 2026-09-30. Research information, not medical advice.

Genes linked to Iodotyrosyl coupling defect

Where Iodotyrosyl coupling defect variants cluster

Known disease-causing variants in Iodotyrosyl coupling defect

VariantPositionProtein partClinical label
TG G2375R2375Cholinesterase-like (ChEL)Disease-causing (★★)
TG R2336Q2336Cholinesterase-like (ChEL)Disease-causing (★★)
TG R2242H2242Cholinesterase-like (ChEL)Disease-causing (★★)
TG C1728R1728Type IIIBDisease-causing (★)
TG C1996S1996Type IIIBDisease-causing
TG C2264Y2264Cholinesterase-like (ChEL)Disease-causing
TG C1077R1077Thyroglobulin type-1 9Disease-causing
TG C1245R1245Disease-causing

Which prediction tools work for Iodotyrosyl coupling defect

How often each tool ranks a disease-causing variant above a harmless one (AUROC × 100).

Diseases related to Iodotyrosyl coupling defect

Frequently asked questions

Which genes are linked to Iodotyrosyl coupling defect?

In CATVariant, Iodotyrosyl coupling defect is linked to 1 analyzed protein: TG (Thyroglobulin).

How many genetic variants are linked to Iodotyrosyl coupling defect?

193 variants: 8 are classified as disease-causing (pathogenic or likely pathogenic) in ClinVar and 158 are of uncertain significance or have conflicting reports.

Which uncertain variants in Iodotyrosyl coupling defect look disease-causing?

None of the uncertain variants currently reaches the likely-pathogenic range on computable evidence alone.

About this data

Variant–disease links come from ClinVar, Open Targets and UniProt, pooled from the latest public CATVariant analysis of each human protein. Evidence scores use the ACMG/AMP Bayesian points scale with computable criteria only (position among known disease variants, rarity in gnomAD, calibrated predictors, deep mutational scanning); there is no family or patient data, so they prioritise variants for expert review and never classify them.

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