Immunodeficiency, common variable, 10: genes and variants

Immunodeficiency, common variable, 10 is linked to 6 analyzed proteins (TNFRSF13B, NFKB2, TNFRSF13C, CD19, ICOS and NFKB1). 5 DNA variants are known to cause it; 574 more are uncertain, and 0 of those already look disease-causing on computable evidence.

Last updated 2026-09-30. Research information, not medical advice.

Also known as: immunodeficiency, common variable, 1; Immunodeficiency, common variable, 12; immunodeficiency, common variable, 2; immunodeficiency, common variable, 3; immunodeficiency, common variable, 4; Immunodeficiency, common variable, 5

Genes linked to Immunodeficiency, common variable, 10

Weakly linked (only a few uncertain records): MS4A1.

Known disease-causing variants in Immunodeficiency, common variable, 10

VariantPositionProtein partClinical label
NFKB2 D865G865Disease-causing (★★)
TNFRSF13C A52T52ExtracellularDisease-causing (★)
TNFRSF13B C104Y104TNFR-Cys 2Disease-causing
CD19 W52C52Ig-like C2-type 1Disease-causing
TNFRSF13B A181E181TransmembraneDisease-causing

Diseases related to Immunodeficiency, common variable, 10

Frequently asked questions

Which genes are linked to Immunodeficiency, common variable, 10?

In CATVariant, Immunodeficiency, common variable, 10 is linked to 6 analyzed proteins: TNFRSF13B (Tumor necrosis factor receptor superfamily member 13B), NFKB2 (Nuclear factor NF-kappa-B p100 subunit), TNFRSF13C (Tumor necrosis factor receptor superfamily member 13C), CD19 (B-lymphocyte antigen CD19), ICOS (Inducible T-cell costimulator) and NFKB1 (Nuclear factor NF-kappa-B p105 subunit).

How many genetic variants are linked to Immunodeficiency, common variable, 10?

642 variants: 5 are classified as disease-causing (pathogenic or likely pathogenic) in ClinVar and 574 are of uncertain significance or have conflicting reports.

Which uncertain variants in Immunodeficiency, common variable, 10 look disease-causing?

None of the uncertain variants currently reaches the likely-pathogenic range on computable evidence alone.

About this data

Variant–disease links come from ClinVar, Open Targets and UniProt, pooled from the latest public CATVariant analysis of each human protein. Evidence scores use the ACMG/AMP Bayesian points scale with computable criteria only (position among known disease variants, rarity in gnomAD, calibrated predictors, deep mutational scanning); there is no family or patient data, so they prioritise variants for expert review and never classify them.

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