Growth hormone insensitivity with immune dysregulation 1, autosomal recessive: genes and variants

Growth hormone insensitivity with immune dysregulation 1, autosomal recessive is linked to 1 analyzed protein (STAT5B). 3 DNA variants are known to cause it; 179 more are uncertain, and 0 of those already look disease-causing on computable evidence.

Last updated 2026-09-30. Research information, not medical advice.

Genes linked to Growth hormone insensitivity with immune dysregulation 1, autosomal recessive

Known disease-causing variants in Growth hormone insensitivity with immune dysregulation 1, autosomal recessive

VariantPositionProtein partClinical label
STAT5B Q177P177Disease-causing
STAT5B Q474R474Disease-causing
STAT5B A630P630SH2Disease-causing

Diseases related to Growth hormone insensitivity with immune dysregulation 1, autosomal recessive

Frequently asked questions

Which genes are linked to Growth hormone insensitivity with immune dysregulation 1, autosomal recessive?

In CATVariant, Growth hormone insensitivity with immune dysregulation 1, autosomal recessive is linked to 1 analyzed protein: STAT5B (Signal transducer and activator of transcription 5B).

How many genetic variants are linked to Growth hormone insensitivity with immune dysregulation 1, autosomal recessive?

198 variants: 3 are classified as disease-causing (pathogenic or likely pathogenic) in ClinVar and 179 are of uncertain significance or have conflicting reports.

Which uncertain variants in Growth hormone insensitivity with immune dysregulation 1, autosomal recessive look disease-causing?

None of the uncertain variants currently reaches the likely-pathogenic range on computable evidence alone.

About this data

Variant–disease links come from ClinVar, Open Targets and UniProt, pooled from the latest public CATVariant analysis of each human protein. Evidence scores use the ACMG/AMP Bayesian points scale with computable criteria only (position among known disease variants, rarity in gnomAD, calibrated predictors, deep mutational scanning); there is no family or patient data, so they prioritise variants for expert review and never classify them.

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