GATA binding protein 1 related thrombocytopenia with dyserythropoiesis: genes and variants

GATA binding protein 1 related thrombocytopenia with dyserythropoiesis is linked to 1 analyzed protein (GATA1). 2 DNA variants are known to cause it; 134 more are uncertain, and 0 of those already look disease-causing on computable evidence.

Last updated 2026-09-30. Research information, not medical advice.

Genes linked to GATA binding protein 1 related thrombocytopenia with dyserythropoiesis

Known disease-causing variants in GATA binding protein 1 related thrombocytopenia with dyserythropoiesis

VariantPositionProtein partClinical label
GATA1 T263M263GATA-type 2Disease-causing (★)
GATA1 M1I1Disease-causing (★)

Diseases related to GATA binding protein 1 related thrombocytopenia with dyserythropoiesis

Frequently asked questions

Which genes are linked to GATA binding protein 1 related thrombocytopenia with dyserythropoiesis?

In CATVariant, GATA binding protein 1 related thrombocytopenia with dyserythropoiesis is linked to 1 analyzed protein: GATA1 (Erythroid transcription factor).

How many genetic variants are linked to GATA binding protein 1 related thrombocytopenia with dyserythropoiesis?

159 variants: 2 are classified as disease-causing (pathogenic or likely pathogenic) in ClinVar and 134 are of uncertain significance or have conflicting reports.

Which uncertain variants in GATA binding protein 1 related thrombocytopenia with dyserythropoiesis look disease-causing?

None of the uncertain variants currently reaches the likely-pathogenic range on computable evidence alone.

About this data

Variant–disease links come from ClinVar, Open Targets and UniProt, pooled from the latest public CATVariant analysis of each human protein. Evidence scores use the ACMG/AMP Bayesian points scale with computable criteria only (position among known disease variants, rarity in gnomAD, calibrated predictors, deep mutational scanning); there is no family or patient data, so they prioritise variants for expert review and never classify them.

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