FGFR3-related chondrodysplasia: genes and variants

FGFR3-related chondrodysplasia is linked to 1 analyzed protein (FGFR3). 20 DNA variants are known to cause it; 107 more are uncertain, and 5 of those already look disease-causing on computable evidence.

Last updated 2026-09-30. Research information, not medical advice.

Genes linked to FGFR3-related chondrodysplasia

Where FGFR3-related chondrodysplasia variants cluster

Known disease-causing variants in FGFR3-related chondrodysplasia

VariantPositionProtein partClinical label
FGFR3 R248C248ExtracellularDisease-causing (★★★★)
FGFR3 N540D540Protein kinaseDisease-causing (★★)
FGFR3 N540T540Protein kinaseDisease-causing (★★)
FGFR3 N540S540Protein kinaseDisease-causing (★★)
FGFR3 N540K540Protein kinaseDisease-causing (★★)
FGFR3 K650Q650Protein kinaseDisease-causing (★★)
FGFR3 K650N650Protein kinaseDisease-causing (★★)
FGFR3 R621H621Protein kinaseDisease-causing (★★)
FGFR3 S249C249ExtracellularDisease-causing (★★)
FGFR3 Y278C278Ig-like C2-type 3Disease-causing (★★)
FGFR3 S279C279Ig-like C2-type 3Disease-causing (★★)
FGFR3 S344C344Ig-like C2-type 3Disease-causing (★★)
FGFR3 S348C348Ig-like C2-type 3Disease-causing (★★)
FGFR3 Y373C373ExtracellularDisease-causing (★★)
FGFR3 G375C375ExtracellularDisease-causing (★★)
FGFR3 G380R380TransmembraneDisease-causing (★★)
FGFR3 S371C371ExtracellularDisease-causing (★★)
FGFR3 N428S428CytoplasmicDisease-causing (★★)
FGFR3 T546K546Protein kinaseDisease-causing (★)
FGFR3 P550H550Protein kinaseDisease-causing (★)

Uncertain variants in FGFR3-related chondrodysplasia that look disease-causing

VariantPositionProtein partClinical labelEvidence
FGFR3 S249Y249ExtracellularConflicting reports (★)+7: 2 other pathogenic changes within 3 positions; S249C at the same position is pathogenic; seen in 4.8e-06 of gnomAD DNA copies; REVEL 0.853
FGFR3 R621C621Protein kinaseConflicting reports (★)+6: in a 3D region that tolerates change poorly (1A); R621H at the same position is pathogenic; not seen in the gnomAD population database; AlphaMissense 1.00
FGFR3 S249F249ExtracellularConflicting reports (★)+6: 2 other pathogenic changes within 3 positions; S249C at the same position is pathogenic; REVEL 0.817
FGFR3 R621L621Protein kinaseUncertain (★★)+6: in a 3D region that tolerates change poorly (1A); R621H at the same position is pathogenic; not seen in the gnomAD population database; AlphaMissense 1.00
FGFR3 K650E650Protein kinaseUncertain (★)+6: 2 other pathogenic changes within 3 positions; K650Q at the same position is pathogenic; not seen in the gnomAD population database; AlphaMissense 0.98

Which prediction tools work for FGFR3-related chondrodysplasia

How often each tool ranks a disease-causing variant above a harmless one (AUROC × 100).

Same protein, different disease

Diseases related to FGFR3-related chondrodysplasia

Frequently asked questions

Which genes are linked to FGFR3-related chondrodysplasia?

In CATVariant, FGFR3-related chondrodysplasia is linked to 1 analyzed protein: FGFR3 (Fibroblast growth factor receptor 3).

How many genetic variants are linked to FGFR3-related chondrodysplasia?

135 variants: 20 are classified as disease-causing (pathogenic or likely pathogenic) in ClinVar and 107 are of uncertain significance or have conflicting reports.

Which uncertain variants in FGFR3-related chondrodysplasia look disease-causing?

5 uncertain variants reach the likely-pathogenic range of the ACMG/AMP points scale on computable evidence, for example FGFR3 S249Y, FGFR3 R621C, FGFR3 S249F, FGFR3 R621L and FGFR3 K650E. These are leads for expert review, not diagnoses.

Which variant effect predictor works best for FGFR3-related chondrodysplasia?

Among tools not trained on clinical labels, CADD separates this disease's known disease-causing variants from harmless ones best (AUROC 0.90, based on 10 disease-causing and 25 harmless variants).

About this data

Variant–disease links come from ClinVar, Open Targets and UniProt, pooled from the latest public CATVariant analysis of each human protein. Evidence scores use the ACMG/AMP Bayesian points scale with computable criteria only (position among known disease variants, rarity in gnomAD, calibrated predictors, deep mutational scanning); there is no family or patient data, so they prioritise variants for expert review and never classify them.

Download every variant as CSV · Browse all diseases · Methods · About the Center