Familial visceral amyloidosis, Ostertag type: genes and variants

Familial visceral amyloidosis, Ostertag type is linked to 2 analyzed proteins (FGA and APOA1). 2 DNA variants are known to cause it; 69 more are uncertain, and 0 of those already look disease-causing on computable evidence.

Last updated 2026-09-30. Research information, not medical advice.

Genes linked to Familial visceral amyloidosis, Ostertag type

Known disease-causing variants in Familial visceral amyloidosis, Ostertag type

VariantPositionProtein partClinical label
FGA R573C573Coiled coilDisease-causing (★★)
FGA E545V545Coiled coilDisease-causing (★★)

Diseases related to Familial visceral amyloidosis, Ostertag type

Frequently asked questions

Which genes are linked to Familial visceral amyloidosis, Ostertag type?

In CATVariant, Familial visceral amyloidosis, Ostertag type is linked to 2 analyzed proteins: FGA (Fibrinogen alpha chain) and APOA1 (Apolipoprotein A-I).

How many genetic variants are linked to Familial visceral amyloidosis, Ostertag type?

74 variants: 2 are classified as disease-causing (pathogenic or likely pathogenic) in ClinVar and 69 are of uncertain significance or have conflicting reports.

Which uncertain variants in Familial visceral amyloidosis, Ostertag type look disease-causing?

None of the uncertain variants currently reaches the likely-pathogenic range on computable evidence alone.

About this data

Variant–disease links come from ClinVar, Open Targets and UniProt, pooled from the latest public CATVariant analysis of each human protein. Evidence scores use the ACMG/AMP Bayesian points scale with computable criteria only (position among known disease variants, rarity in gnomAD, calibrated predictors, deep mutational scanning); there is no family or patient data, so they prioritise variants for expert review and never classify them.

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