Familial hyperparathyroidism or Hypocalciuric hypercalcaemia: genes and variants

Familial hyperparathyroidism or Hypocalciuric hypercalcaemia is linked to 2 analyzed proteins (CASR and RET). 5 DNA variants are known to cause it; 20 more are uncertain, and 0 of those already look disease-causing on computable evidence.

Last updated 2026-09-30. Research information, not medical advice.

Genes linked to Familial hyperparathyroidism or Hypocalciuric hypercalcaemia

Weakly linked (only a few uncertain records): CDC73 and CDKN1B.

Where Familial hyperparathyroidism or Hypocalciuric hypercalcaemia variants cluster

Known disease-causing variants in Familial hyperparathyroidism or Hypocalciuric hypercalcaemia

VariantPositionProtein partClinical label
CASR T138M138Ligand-binding 1 (LB1)Disease-causing (★★)
CASR S147L147Ligand-binding 1 (LB1)Disease-causing (★★)
CASR G553R553Cysteine-rich (CR)Disease-causing (★★)
CASR F183L183Ligand-binding 1 (LB1)Disease-causing (★)
CASR S171R171Ligand-binding 1 (LB1)Disease-causing (★)

Same protein, different disease

Diseases related to Familial hyperparathyroidism or Hypocalciuric hypercalcaemia

Frequently asked questions

Which genes are linked to Familial hyperparathyroidism or Hypocalciuric hypercalcaemia?

In CATVariant, Familial hyperparathyroidism or Hypocalciuric hypercalcaemia is linked to 2 analyzed proteins: CASR (Extracellular calcium-sensing receptor) and RET (Proto-oncogene tyrosine-protein kinase receptor Ret).

How many genetic variants are linked to Familial hyperparathyroidism or Hypocalciuric hypercalcaemia?

25 variants: 5 are classified as disease-causing (pathogenic or likely pathogenic) in ClinVar and 20 are of uncertain significance or have conflicting reports.

Which uncertain variants in Familial hyperparathyroidism or Hypocalciuric hypercalcaemia look disease-causing?

None of the uncertain variants currently reaches the likely-pathogenic range on computable evidence alone.

About this data

Variant–disease links come from ClinVar, Open Targets and UniProt, pooled from the latest public CATVariant analysis of each human protein. Evidence scores use the ACMG/AMP Bayesian points scale with computable criteria only (position among known disease variants, rarity in gnomAD, calibrated predictors, deep mutational scanning); there is no family or patient data, so they prioritise variants for expert review and never classify them.

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