Epidermolytic palmoplantar keratoderma, 1: genes and variants

Epidermolytic palmoplantar keratoderma, 1 is linked to 3 analyzed proteins (KRT9, KRT1 and KRT10). 12 DNA variants are known to cause it; 7 more are uncertain, and 0 of those already look disease-causing on computable evidence.

Last updated 2026-09-30. Research information, not medical advice.

Genes linked to Epidermolytic palmoplantar keratoderma, 1

Where Epidermolytic palmoplantar keratoderma, 1 variants cluster

Known disease-causing variants in Epidermolytic palmoplantar keratoderma, 1

VariantPositionProtein partClinical label
KRT9 R163Q163IF rodDisease-causing (★★★★)
KRT9 N161S161IF rodDisease-causing (★★)
KRT9 N161K161IF rodDisease-causing (★★)
KRT9 R163W163IF rodDisease-causing (★★)
KRT9 R163L163IF rodDisease-causing (★★)
KRT9 L160V160IF rodDisease-causing
KRT9 L160F160IF rodDisease-causing
KRT9 N161Y161IF rodDisease-causing
KRT9 N161I161IF rodDisease-causing
KRT9 V171M171IF rodDisease-causing
KRT9 L168S168IF rodDisease-causing
KRT9 Q172P172IF rodDisease-causing

Which prediction tools work for Epidermolytic palmoplantar keratoderma, 1

How often each tool ranks a disease-causing variant above a harmless one (AUROC × 100).

Diseases related to Epidermolytic palmoplantar keratoderma, 1

Frequently asked questions

Which genes are linked to Epidermolytic palmoplantar keratoderma, 1?

In CATVariant, Epidermolytic palmoplantar keratoderma, 1 is linked to 3 analyzed proteins: KRT9 (Keratin, type I cytoskeletal 9), KRT1 (Keratin, type II cytoskeletal 1) and KRT10 (Keratin, type I cytoskeletal 10).

How many genetic variants are linked to Epidermolytic palmoplantar keratoderma, 1?

23 variants: 12 are classified as disease-causing (pathogenic or likely pathogenic) in ClinVar and 7 are of uncertain significance or have conflicting reports.

Which uncertain variants in Epidermolytic palmoplantar keratoderma, 1 look disease-causing?

None of the uncertain variants currently reaches the likely-pathogenic range on computable evidence alone.

About this data

Variant–disease links come from ClinVar, Open Targets and UniProt, pooled from the latest public CATVariant analysis of each human protein. Evidence scores use the ACMG/AMP Bayesian points scale with computable criteria only (position among known disease variants, rarity in gnomAD, calibrated predictors, deep mutational scanning); there is no family or patient data, so they prioritise variants for expert review and never classify them.

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