Epidermolysis bullosa simplex 1D, generalized, intermediate or severe, autosomal recessive: genes and variants

Epidermolysis bullosa simplex 1D, generalized, intermediate or severe, autosomal recessive is linked to 2 analyzed proteins (KRT14 and KRT5). 5 DNA variants are known to cause it; 1 more are uncertain, and 0 of those already look disease-causing on computable evidence.

Last updated 2026-09-30. Research information, not medical advice.

Genes linked to Epidermolysis bullosa simplex 1D, generalized, intermediate or severe, autosomal recessive

Known disease-causing variants in Epidermolysis bullosa simplex 1D, generalized, intermediate or severe, autosomal recessive

VariantPositionProtein partClinical label
KRT14 M272T272IF rodDisease-causing (★★)
KRT14 R388H388IF rodDisease-causing (★★)
KRT5 M327T327IF rodDisease-causing (★★)
KRT14 R125P125IF rodDisease-causing (★)
KRT14 E144A144IF rodDisease-causing

Same protein, different disease

Diseases related to Epidermolysis bullosa simplex 1D, generalized, intermediate or severe, autosomal recessive

Frequently asked questions

Which genes are linked to Epidermolysis bullosa simplex 1D, generalized, intermediate or severe, autosomal recessive?

In CATVariant, Epidermolysis bullosa simplex 1D, generalized, intermediate or severe, autosomal recessive is linked to 2 analyzed proteins: KRT14 (Keratin, type I cytoskeletal 14) and KRT5 (Keratin, type II cytoskeletal 5).

How many genetic variants are linked to Epidermolysis bullosa simplex 1D, generalized, intermediate or severe, autosomal recessive?

15 variants: 5 are classified as disease-causing (pathogenic or likely pathogenic) in ClinVar and 1 are of uncertain significance or have conflicting reports.

Which uncertain variants in Epidermolysis bullosa simplex 1D, generalized, intermediate or severe, autosomal recessive look disease-causing?

None of the uncertain variants currently reaches the likely-pathogenic range on computable evidence alone.

About this data

Variant–disease links come from ClinVar, Open Targets and UniProt, pooled from the latest public CATVariant analysis of each human protein. Evidence scores use the ACMG/AMP Bayesian points scale with computable criteria only (position among known disease variants, rarity in gnomAD, calibrated predictors, deep mutational scanning); there is no family or patient data, so they prioritise variants for expert review and never classify them.

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