Enhanced S-cone syndrome: genes and variants

Enhanced S-cone syndrome is linked to 1 analyzed protein (NR2E3). 12 DNA variants are known to cause it; 44 more are uncertain, and 0 of those already look disease-causing on computable evidence.

Last updated 2026-09-30. Research information, not medical advice.

Also known as: ENHANCED S-CONE SYNDROME 1

Genes linked to Enhanced S-cone syndrome

Where Enhanced S-cone syndrome variants cluster

Known disease-causing variants in Enhanced S-cone syndrome

VariantPositionProtein partClinical label
NR2E3 R309G309NR LBDDisease-causing (★★)
NR2E3 R311Q311NR LBDDisease-causing (★★)
NR2E3 R76Q76Nuclear receptorDisease-causing (★★)
NR2E3 Y81C81Nuclear receptorDisease-causing (★★)
NR2E3 R97H97Nuclear receptorDisease-causing (★★)
NR2E3 A102D102Nuclear receptorDisease-causing (★★)
NR2E3 R104W104Nuclear receptorDisease-causing (★★)
NR2E3 V118M118Nuclear receptorDisease-causing (★★)
NR2E3 G216S216NR LBDDisease-causing (★★)
NR2E3 V342A342NR LBDDisease-causing (★★)
NR2E3 M407K407NR LBDDisease-causing (★★)
NR2E3 R309L309NR LBDDisease-causing (★)

Which prediction tools work for Enhanced S-cone syndrome

How often each tool ranks a disease-causing variant above a harmless one (AUROC × 100).

Same protein, different disease

Diseases related to Enhanced S-cone syndrome

Frequently asked questions

Which genes are linked to Enhanced S-cone syndrome?

In CATVariant, Enhanced S-cone syndrome is linked to 1 analyzed protein: NR2E3 (Photoreceptor-specific nuclear receptor).

How many genetic variants are linked to Enhanced S-cone syndrome?

78 variants: 12 are classified as disease-causing (pathogenic or likely pathogenic) in ClinVar and 44 are of uncertain significance or have conflicting reports.

Which uncertain variants in Enhanced S-cone syndrome look disease-causing?

None of the uncertain variants currently reaches the likely-pathogenic range on computable evidence alone.

Which variant effect predictor works best for Enhanced S-cone syndrome?

Among tools not trained on clinical labels, CADD separates this disease's known disease-causing variants from harmless ones best (AUROC 0.79, based on 11 disease-causing and 9 harmless variants).

About this data

Variant–disease links come from ClinVar, Open Targets and UniProt, pooled from the latest public CATVariant analysis of each human protein. Evidence scores use the ACMG/AMP Bayesian points scale with computable criteria only (position among known disease variants, rarity in gnomAD, calibrated predictors, deep mutational scanning); there is no family or patient data, so they prioritise variants for expert review and never classify them.

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