Crigler-Najjar syndrome: genes and variants

Crigler-Najjar syndrome is linked to 1 analyzed protein (UGT1A1). 14 DNA variants are known to cause it; 38 more are uncertain, and 0 of those already look disease-causing on computable evidence.

Last updated 2026-09-30. Research information, not medical advice.

Also known as: Crigler-Najjar syndrome type 1; Crigler-Najjar syndrome type 2; Crigler-Najjar syndrome, type II

Genes linked to Crigler-Najjar syndrome

Known disease-causing variants in Crigler-Najjar syndrome

VariantPositionProtein partClinical label
UGT1A1 R336W336Disease-causing (★★)
UGT1A1 R209W209Disease-causing (★★)
UGT1A1 R336Q336Disease-causing (★★)
UGT1A1 P387R387Disease-causing (★★)
UGT1A1 Y486D486Disease-causing (★★)
UGT1A1 Q331R331Disease-causing (★★)
UGT1A1 Q357R357Disease-causing (★★)
UGT1A1 S375F375Disease-causing (★★)
UGT1A1 G276R276Disease-causing (★)
UGT1A1 L443P443Disease-causing (★)
UGT1A1 G395V395Disease-causing (★)
UGT1A1 L15R15Disease-causing (★)
UGT1A1 F83L83Disease-causing (★)
UGT1A1 R450L450Disease-causing (★)

Same protein, different disease

Diseases related to Crigler-Najjar syndrome

Frequently asked questions

Which genes are linked to Crigler-Najjar syndrome?

In CATVariant, Crigler-Najjar syndrome is linked to 1 analyzed protein: UGT1A1 (UDP-glucuronosyltransferase 1A1).

How many genetic variants are linked to Crigler-Najjar syndrome?

83 variants: 14 are classified as disease-causing (pathogenic or likely pathogenic) in ClinVar and 38 are of uncertain significance or have conflicting reports.

Which uncertain variants in Crigler-Najjar syndrome look disease-causing?

None of the uncertain variants currently reaches the likely-pathogenic range on computable evidence alone.

About this data

Variant–disease links come from ClinVar, Open Targets and UniProt, pooled from the latest public CATVariant analysis of each human protein. Evidence scores use the ACMG/AMP Bayesian points scale with computable criteria only (position among known disease variants, rarity in gnomAD, calibrated predictors, deep mutational scanning); there is no family or patient data, so they prioritise variants for expert review and never classify them.

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