Corneal dystrophy, Meesmann, 2: genes and variants

Corneal dystrophy, Meesmann, 2 is linked to 2 analyzed proteins (KRT3 and KRT12). 11 DNA variants are known to cause it; 6 more are uncertain, and 0 of those already look disease-causing on computable evidence.

Last updated 2026-09-30. Research information, not medical advice.

Also known as: Corneal dystrophy, Meesmann, 1

Genes linked to Corneal dystrophy, Meesmann, 2

Where Corneal dystrophy, Meesmann, 2 variants cluster

Known disease-causing variants in Corneal dystrophy, Meesmann, 2

VariantPositionProtein partClinical label
KRT3 E498K498IF rodDisease-causing (★)
KRT3 E509D509IF rodDisease-causing (★)
KRT12 R135G135IF rodDisease-causing
KRT12 R135I135IF rodDisease-causing
KRT12 R135T135IF rodDisease-causing
KRT3 E498V498IF rodDisease-causing
KRT12 L132P132IF rodDisease-causing
KRT12 V143L143IF rodDisease-causing
KRT3 R503P503IF rodDisease-causing
KRT12 L140R140IF rodDisease-causing
KRT12 Y429D429IF rodDisease-causing

Which prediction tools work for Corneal dystrophy, Meesmann, 2

How often each tool ranks a disease-causing variant above a harmless one (AUROC × 100).

Frequently asked questions

Which genes are linked to Corneal dystrophy, Meesmann, 2?

In CATVariant, Corneal dystrophy, Meesmann, 2 is linked to 2 analyzed proteins: KRT3 (Keratin, type II cytoskeletal 3) and KRT12 (Keratin, type I cytoskeletal 12).

How many genetic variants are linked to Corneal dystrophy, Meesmann, 2?

19 variants: 11 are classified as disease-causing (pathogenic or likely pathogenic) in ClinVar and 6 are of uncertain significance or have conflicting reports.

Which uncertain variants in Corneal dystrophy, Meesmann, 2 look disease-causing?

None of the uncertain variants currently reaches the likely-pathogenic range on computable evidence alone.

About this data

Variant–disease links come from ClinVar, Open Targets and UniProt, pooled from the latest public CATVariant analysis of each human protein. Evidence scores use the ACMG/AMP Bayesian points scale with computable criteria only (position among known disease variants, rarity in gnomAD, calibrated predictors, deep mutational scanning); there is no family or patient data, so they prioritise variants for expert review and never classify them.

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