Cenani-Lenz syndactyly syndrome: genes and variants

Cenani-Lenz syndactyly syndrome is linked to 1 analyzed protein (LRP4). 9 DNA variants are known to cause it; 494 more are uncertain, and 0 of those already look disease-causing on computable evidence.

Last updated 2026-09-30. Research information, not medical advice.

Genes linked to Cenani-Lenz syndactyly syndrome

Known disease-causing variants in Cenani-Lenz syndactyly syndrome

VariantPositionProtein partClinical label
LRP4 E1233A1233LDL-receptor class B 14Disease-causing (★★)
LRP4 D137N137LDL-receptor class A 3Disease-causing (★)
LRP4 D300N300LDL-receptor class A 7Disease-causing (★)
LRP4 R632C632LDL-receptor class B 4Disease-causing (★)
LRP4 D99H99LDL-receptor class A 2Disease-causing (★)
LRP4 C160Y160LDL-receptor class A 4Disease-causing
LRP4 D449N449ExtracellularDisease-causing
LRP4 D529N529LDL-receptor class B 2Disease-causing
LRP4 T461P461ExtracellularDisease-causing

Which prediction tools work for Cenani-Lenz syndactyly syndrome

How often each tool ranks a disease-causing variant above a harmless one (AUROC × 100).

Diseases related to Cenani-Lenz syndactyly syndrome

Frequently asked questions

Which genes are linked to Cenani-Lenz syndactyly syndrome?

In CATVariant, Cenani-Lenz syndactyly syndrome is linked to 1 analyzed protein: LRP4 (Low-density lipoprotein receptor-related protein 4).

How many genetic variants are linked to Cenani-Lenz syndactyly syndrome?

528 variants: 9 are classified as disease-causing (pathogenic or likely pathogenic) in ClinVar and 494 are of uncertain significance or have conflicting reports.

Which uncertain variants in Cenani-Lenz syndactyly syndrome look disease-causing?

None of the uncertain variants currently reaches the likely-pathogenic range on computable evidence alone.

Which variant effect predictor works best for Cenani-Lenz syndactyly syndrome?

Among tools not trained on clinical labels, AlphaMissense separates this disease's known disease-causing variants from harmless ones best (AUROC 0.98, based on 8 disease-causing and 13 harmless variants).

About this data

Variant–disease links come from ClinVar, Open Targets and UniProt, pooled from the latest public CATVariant analysis of each human protein. Evidence scores use the ACMG/AMP Bayesian points scale with computable criteria only (position among known disease variants, rarity in gnomAD, calibrated predictors, deep mutational scanning); there is no family or patient data, so they prioritise variants for expert review and never classify them.

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