Bloom syndrome: genes and variants

Bloom syndrome is linked to 1 analyzed protein (BLM). 9 DNA variants are known to cause it; 1,736 more are uncertain, and 1 of those already look disease-causing on computable evidence.

Last updated 2026-09-30. Research information, not medical advice.

Genes linked to Bloom syndrome

Known disease-causing variants in Bloom syndrome

VariantPositionProtein partClinical label
BLM Q672R672Disease-causing (★★)
BLM C1055S1055Disease-causing (★★)
BLM C1055R1055Disease-causing (★★)
BLM C901Y901Helicase C-terminalDisease-causing (★★)
BLM D1064V1064Disease-causing (★★)
BLM M814K814Helicase ATP-bindingDisease-causing
BLM M1L1Disease-causing
BLM G952A952Helicase C-terminalDisease-causing
BLM C1036F1036Disease-causing

Uncertain variants in Bloom syndrome that look disease-causing

VariantPositionProtein partClinical labelEvidence
BLM C1055Y1055Conflicting reports (★)+7: 2 other pathogenic changes within 3 positions; C1055S at the same position is pathogenic; seen in 1.4e-06 of gnomAD DNA copies; REVEL 0.851

Diseases related to Bloom syndrome

Frequently asked questions

Which genes are linked to Bloom syndrome?

In CATVariant, Bloom syndrome is linked to 1 analyzed protein: BLM (RecQ-like DNA helicase BLM).

How many genetic variants are linked to Bloom syndrome?

1,813 variants: 9 are classified as disease-causing (pathogenic or likely pathogenic) in ClinVar and 1,736 are of uncertain significance or have conflicting reports.

Which uncertain variants in Bloom syndrome look disease-causing?

1 uncertain variants reach the likely-pathogenic range of the ACMG/AMP points scale on computable evidence, for example BLM C1055Y. These are leads for expert review, not diagnoses.

About this data

Variant–disease links come from ClinVar, Open Targets and UniProt, pooled from the latest public CATVariant analysis of each human protein. Evidence scores use the ACMG/AMP Bayesian points scale with computable criteria only (position among known disease variants, rarity in gnomAD, calibrated predictors, deep mutational scanning); there is no family or patient data, so they prioritise variants for expert review and never classify them.

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