17-alpha-hydroxylase/17,20-lyase deficiency, combined complete: genes and variants

17-alpha-hydroxylase/17,20-lyase deficiency, combined complete is linked to 1 analyzed protein (CYP17A1). 3 DNA variants are known to cause it; 0 more are uncertain, and 0 of those already look disease-causing on computable evidence.

Last updated 2026-09-30. Research information, not medical advice.

Genes linked to 17-alpha-hydroxylase/17,20-lyase deficiency, combined complete

Known disease-causing variants in 17-alpha-hydroxylase/17,20-lyase deficiency, combined complete

VariantPositionProtein partClinical label
CYP17A1 F93C93Disease-causing
CYP17A1 F114V114Disease-causing
CYP17A1 Y329D329Disease-causing

Same protein, different disease

Diseases related to 17-alpha-hydroxylase/17,20-lyase deficiency, combined complete

Frequently asked questions

Which genes are linked to 17-alpha-hydroxylase/17,20-lyase deficiency, combined complete?

In CATVariant, 17-alpha-hydroxylase/17,20-lyase deficiency, combined complete is linked to 1 analyzed protein: CYP17A1 (Steroid 17-alpha-hydroxylase/17,20 lyase).

How many genetic variants are linked to 17-alpha-hydroxylase/17,20-lyase deficiency, combined complete?

13 variants: 3 are classified as disease-causing (pathogenic or likely pathogenic) in ClinVar and 0 are of uncertain significance or have conflicting reports.

Which uncertain variants in 17-alpha-hydroxylase/17,20-lyase deficiency, combined complete look disease-causing?

None of the uncertain variants currently reaches the likely-pathogenic range on computable evidence alone.

About this data

Variant–disease links come from ClinVar, Open Targets and UniProt, pooled from the latest public CATVariant analysis of each human protein. Evidence scores use the ACMG/AMP Bayesian points scale with computable criteria only (position among known disease variants, rarity in gnomAD, calibrated predictors, deep mutational scanning); there is no family or patient data, so they prioritise variants for expert review and never classify them.

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